GPR35 agonists (YE120, zaprinast, and pamoic acid) promoted wound maintenance in a focus-dependent way independently of cell proliferation, Whilst a particular GPR35 antagonist CID2745687, forskolin, and pertussis toxin reversed the YE120-induced effect. YE120 amplified the mRNA expression of fibronectin and its receptor integrin α5, and ERK1/2 phosphorylation, but these responses https://johne333pak5.59bloggers.com/profile