Max in WT synaptoneurosomes, suggesting that Src signaling may very well be downregulated in KI synapses. Alternatively, our capability to rescue SERT functionality in KI midbrain synaptoneurosomes because of the inhibition of FAK suggests elevated FAK signaling downstream from the Pro32Pro33 mutant, as verified by enhanced pFAK localization in 5-HT https://richarde209iue0.wikiitemization.com/user